DSpace Collection:http://hdl.handle.net/11607/18002024-03-29T12:26:46Z2024-03-29T12:26:46ZPsikiyatrik bulgular ile prezente olan hipoglisemi ile ilişkili korpus kallosum infarktı: olgu sunumuGüler, SibelAygün, Murat Serhathttp://hdl.handle.net/11607/20842016-02-26T01:00:56Z2011-01-01T00:00:00ZTitle: Psikiyatrik bulgular ile prezente olan hipoglisemi ile ilişkili korpus kallosum infarktı: olgu sunumu
Authors: Güler, Sibel; Aygün, Murat Serhat
Abstract: A 54 years old female patient, presenting with psychiatric symptoms, after which corpus callosum infarct was detected is presented with the clinical, laboratory and radiological findings here. The patient, who did not have any psychiatric complaints before was evaluated because of personality changes and sleepy state starting 3 days before and disturbance in gait and speech of one day's duration. Previously she had a diagnosis of diabetes mellitus. Blood glucose was 45 mg/dL. An increase in signal intensity reciprocal to ADC in the corpus callosum was detected in cerebral diffusion magnetic resonance imaging (MRI). The psychiatric findings in this case were considered as “acute and temporary schizoid personality disorder” that may be explained by an underlying organic cause according to DSM-IV. The clinical symptoms that arise from corpus callosum abnormalities are variable. A psychiatric picture develops in approximately one thirds of the cases. On the other hand, corpus callosum infarct presenting with pure psychiatric findings are rarely observed in the literature. This case was considered interesting because of the temporal association between corpus callosum infarct and psychiatric symptoms in the context of coexistent hypoglycemia and left anterior cerebral artery occlusion.2011-01-01T00:00:00ZNon-sirotik portal hipertansiyonun nadir bi nedeni : primer antifosfolipid sendromuUlaş, TurgayBes, CemalGültürk, EmineNamal, EsatPaksoy, FatmaGürbüz, Didem GökçenBorlu, Fatihhttp://hdl.handle.net/11607/20802016-02-26T01:01:00Z2011-01-01T00:00:00ZTitle: Non-sirotik portal hipertansiyonun nadir bi nedeni : primer antifosfolipid sendromu
Authors: Ulaş, Turgay; Bes, Cemal; Gültürk, Emine; Namal, Esat; Paksoy, Fatma; Gürbüz, Didem Gökçen; Borlu, Fatih
Abstract: The antiphospholipid syndrome involves long-term persistence of serum antiphospholipid antibodies and hypercoagulability manifested by venous or arterial thrombosis, recurrent pregnancy loss or thrombocytopenia. Two forms have been described: the “primary syndrome” where there is no evidence of an underlying disease, and the “secondary syndrome”, mainly in the setting of systemic lupus eryhematosus. Classic features of antiphospholipid syndrome include cutaneous signs, such as livedo reticularis, splinter hemorrhages, superficial thrombophlebitis, and leg ulcers; venous thrombotic events, such as cerebral or retinal vein thrombosis; arterial thrombotic events, such as transient attacks or ischemic infarcts, recurrent spontaneous early-term abortions, and thrombocytopenia. Also various hepatic manifestations have been reported with antiphospholipid syndrome in literature, including Budd-Chiari syndrome, hepatic-veno-occlusive disease and occlusion of small hepatic veins, nodular regenerative hyperplasia and portal hypertension. Herein we present a case of non-cirrhotic portal hypertension associated with primary antiphospholipid syndrome which this coexisting is rarely seen in the literature.2011-01-01T00:00:00ZMiller fisher sendromu: bir olgu sunumuGülez, PamirHızarcıoğlu, MuratÜnalp, AycanSiviş, Zuhal Önderhttp://hdl.handle.net/11607/20782016-02-26T01:00:56Z2011-01-01T00:00:00ZTitle: Miller fisher sendromu: bir olgu sunumu
Authors: Gülez, Pamir; Hızarcıoğlu, Murat; Ünalp, Aycan; Siviş, Zuhal Önder
Abstract: Miller-Fisher syndrome is characterized by gait ataxia, external ophtalmoplegia and areflexia and thought as an uncommon variant of Guillain Barre syndrome. Miller-Fisher syndrome is observed in about 5-8% of all Guillain Barre syndrome cases. Acute onset of external ophthalmoplegia is a cardinal feature of Miller-Fisher syndrome. Miller-Fisher syndrome can be distinguished from Guillain Barre syndrome by a few clinical features. Because of the laboratory findings are not spesific for Miller-Fisher syndrome, the diagnosis is made only with suspicion of the disease. Miller-Fisher syndrome was diagnosed at a five years old girl with gait ataxia, ophthalmoplegia, hyporeflexia, cerebrospinal fluid and electromyographic findings. Intravenous immunoglobulin 1g/kg/dose was used for two days. The aim of the case is presented here to remind this very uncommon antity.2011-01-01T00:00:00ZGerçek umblikal kord kisti: olgu sunumuSezer, Selda DemircanKüçük, MertYüksel, HasanOdabaşı, Ali RızaDurmuş, YasinÖzkısacık, SezenTataroğlu, CantenTürkmen, Münevverhttp://hdl.handle.net/11607/20732016-02-26T01:00:58Z2011-01-01T00:00:00ZTitle: Gerçek umblikal kord kisti: olgu sunumu
Authors: Sezer, Selda Demircan; Küçük, Mert; Yüksel, Hasan; Odabaşı, Ali Rıza; Durmuş, Yasin; Özkısacık, Sezen; Tataroğlu, Canten; Türkmen, Münevver
Abstract: Umbilical cord cysts are important due to their association with fetal chromosomal abnormalities and fetal structural defects and in case of their rapid growth of umbilical cord cysts they are associated with compression of umbilical vessels and fetal distress. In this article we aimed to present a case with two umbilical cord cysts detected by ultrasonography at 31 weeks of gestation and who had delivered a healthy male fetus by cesarean section at 37 weeks of gestation. Histopathological examination of placenta and umbilical cord after birth revealed that cysts were true allantoic cysts. An additional urachal pathology and anomaly was not observed and the newborn was discharged after primary excision. As a result, we think that ultrasound examination of umbilical cord should be an important and routine part of obstetric ultrasonography.2011-01-01T00:00:00Z